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The usual process of antigen presentation through the MHC I molecule is based on an interaction between the T-cell receptor and a peptide bound to the MHC class I molecule. The consequences of these polymorphisms are differential susceptibilities to infection and autoimmune diseases that may result from the high diversity of peptides that can bind to MHC class I in different individuals.
Also, MHC class I polymorphisms make it virtually impossible to have a perfect tissue match between donor and recipient, and thus are responsible for graft rejection. MHC class II molecules bind to peptides that are derived from proteins degraded in the endocytic pathway.
Later, the CLIP is exchanged for an antigenic peptide derived from a protein degraded in the endosomal pathway. Different MHC class II alleles can be used as genetic markers for several autoimmune diseases, possibly owing to the peptides that they present. Register Log in.
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Figure 1. The MHC class I antigen-presentation pathway.follow
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Immunological Reactivity of Peptides related to Angiotensin
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